Dabcyl acid, SE
CAS No. 146998-31-4
Dabcyl acid, SE ( Dabcyl, SE )
Catalog No. M23629 CAS No. 146998-31-4
Dabcyl acid, SE is the amino-reactive form of Dabcyl acid and used to prepare a variety of FRET-based probes that contain Dabcyl acid.
Purity : >98% (HPLC)
Size | Price / USD | Stock | Quantity |
50MG | 45 | In Stock |
|
100MG | Get Quote | In Stock |
|
200MG | Get Quote | In Stock |
|
500MG | Get Quote | In Stock |
|
1G | Get Quote | In Stock |
|
Biological Information
-
Product NameDabcyl acid, SE
-
NoteResearch use only, not for human use.
-
Brief DescriptionDabcyl acid, SE is the amino-reactive form of Dabcyl acid and used to prepare a variety of FRET-based probes that contain Dabcyl acid.
-
DescriptionDabcyl acid, SE is the amino-reactive form of Dabcyl acid and used to prepare a variety of FRET-based probes that contain Dabcyl acid.
-
SynonymsDabcyl, SE
-
PathwayOthers
-
TargetOther Targets
-
Recptorothers
-
Research Area——
-
Indication——
Chemical Information
-
CAS Number146998-31-4
-
Formula Weight366.4
-
Molecular FormulaC19H18N4O4
-
Purity>98% (HPLC)
-
SolubilityDMSO:5 mg/mL?(13.64 mM;?Need ultrasonic)
-
SMILESCN(C)c(cc1)ccc1N=Nc(cc1)ccc1C(ON(C(CC1)=O)C1=O)=O
-
Chemical Name——
Shipping & Storage Information
-
Storage(-20℃)
-
ShippingWith Ice Pack
-
Stability≥ 2 years
Reference
-
8-Prenylkaempferol
8-Prenylkaempferol is an effective agent for attenuating pro-inflammatory NO induction, it may be an anti-inflammatory agent for suppressing influenza A virus-induced RANTES production acts by blocking PI3K-mediated transcriptional activation of NF-κB and IRF-3 and in part by interfering with IκB degradation which subsequently decreases NF-κB translocation.
-
Pancreatic Polypepti...
Pancreatic Polypeptide, rat is an agonist of NPY receptor, with high affinity at NPYR4. Pancreatic polypeptide (PP) is a 36-amino acid, C-terminally amidated peptide, and is a member of the PP-fold peptide family. It is produced and secreted by the PP cells of the islets of Langerhans.
-
Crenulatin
Crenulatin has dual- direction effects on apoptosis of cerebral microvascular endothelial cells, inhibitive effect in 25 mg/L and stimulative effect in 100 mg/L group, respectively; the mechanism is related to the alterations of Fas/Bcl-2 expression and caspase-3 activity.